Shade avoidance syndrome (SAS) are traits that plant haveevolved to help sustain light capture and avoid shade by neighboring plants. Thesetraits include enhanced elongation of stems and petioles, upward leaf movement,known as hyponasty, and increased apical dominance. A classical signal toinduce SAS is the reduced ratio between red (R ) and far-red (FR) light, but inaddition to red light sensing, blue light fluence rates can control SASfeatures as well. However, the regulatory network underpinning low-blue-inducedelongation growth is less known. The authors of the paper studied the hormonalcontrol of low-blue-induced hypocotyl elongation, and showed that auxintransport and signaling control this response. But they also found thatinhibition of auxin pathways don’t inhibit completely the response,demonstrating that other component is also controlling this plant features. Theyfound that brassinosteroids (BRs) also regulate elongation response to low bluelight, and the combined action of both hormones mainly control this shadeavoidance response, much probably by regulation of specific XTH genes, that aremembers of a cell wall modifying protein family.